Abnormal Aspartyl Residues and Amyloid Formation

Steven Clarke, PhD
Regents of the University of California (Los Angeles, CA)
Year Awarded:
Grant Duration:
April 1, 1995 to March 31, 1998
Alzheimer's Disease
Award Amount:
Grant Reference ID:
Award Type:
Award Region:
US Southwestern

Abnormal Aspartyl Residues and Amyloid Formation



[Book Chapter] Perna, A.F., D'Aniello, A., Lowenson, J.D., Clarke, S., et al. (1997) D-Aspartate content of erythrocyte membrane proteins is decreased. In: Uremia: Implications for the Repair of Damaged Proteins. J. Amer. Soc. Nephrol. 8:95-104.

Roher, A.E., Lowenson, J.D., Clarke, S., Woods, A.S., et al. (1997) Beta-amyloid (1-42) is a major component of cerebrovascular amyloid deposits: implications for the pathology of Alzheimer's disease. Proc. Natl. Acad. Sci. USA. 90:10836-10840.  

[Book Chapter] Lowenson, J.D. and Clarke, S. (1995) Recognition of isomerized and racemized aspartyl residues in peptides by the protein L-isoaspartate(D-Aspartate) O-methyltransferase. In: Deamidation and Isoaspartate Formation in Peptides and Proteins. Aswad, D. W., ed. CRC Press, Boca Raton, FL, pp. 47-64.

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